Myoblast transfer therapy for relieving pain and for...

Drug – bio-affecting and body treating compositions – Whole live micro-organism – cell – or virus containing – Genetically modified micro-organism – cell – or virus

Reexamination Certificate

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C435S366000, C424S093200

Reexamination Certificate

active

09986344

ABSTRACT:
An analgesic benefit is realized by continuously supplying a peptide in vivo that activates an opioid receptor or that interferes with the binding of substance P to its receptors. The long-term, continuous provision of such a peptide can be accomplished by (a) transducing myogenic cells with DNA expressing the peptide and (b) administering the transduced myogenic cells to a patient, such that the cells continuously produce the peptide.

REFERENCES:
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patent: WO 92/16547 (1992-10-01), None
patent: WO 96/18303 (1996-06-01), None
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Beutler et al. “Retrovirus-mediated expression of an artificial beta-endorphin precursor in primary fibroblasts,” J. Neurochem. 64(2): 475-481, 1995.
Skuk, D., “Myoblast transplantaion for inherited myopathies: a clinical approach,” Expert Opin. Biol. Ther. 4 (12): 1871-1875, 2004.
Satoh et al., Myotubes can be formed within implanted adipose tissue, Transplant. Proc. 24(6): 3017-3019, 1992.
Takeuchi, T., et al., “Production of a Therapeutic Peptide Enkephalin from a Variety of Non-Endocrine Cell Lines Using a Novel Expression Vector for Fusion Peptides,” Gene Therapy, vol. 2, pp. 689 (1995).
Sandra, A., “Reversal by Insulin of Concanavalin A Inhibition of Myotube Formation and Evidence for Binding Sites,” Endocrinology, vol. 105, No. 2, pp. 391-401 (Aug. 1979).
Wu, et al., “Implantation of AtT-20 or Genetically Modified AIT-20/hENK Cells in Mouse Spinal Cord Induced Antinociception and Opioid Tolerance,” Journal of Neuroscience, vol. 14, No. 8, pp. 4806-4814 (1994).

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