Methods and compositions for treatment of...

Drug – bio-affecting and body treating compositions – Designated organic active ingredient containing – Radical -xh acid – or anhydride – acid halide or salt thereof...

Reexamination Certificate

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C514S566000

Reexamination Certificate

active

10617943

ABSTRACT:
This invention relates to the methods and pharmaceutical compositions for treating diseases or disorders associated with oxidative stress and/or genomic instability. In particular, the invention relates to methods for treating ataxia-telangeictasia (AT) and such disease states by administering a therapeutically effective amount of a chelating agent to increase genomic stability and/or decrease oxidative stress.

REFERENCES:
Shackelford et al. Desferrioxamine treatment increases the genomic stability ofAtaxia-telangiectasiacells, DNA Repair, Sep. 18, 2003, vol. 2, pp. 971-981.
Weil et al., Radiation Induces Genomic Instability and Mammary Ductal Dysplasia in ATM Heterozgous Mice, 2001, Oncogene, vol. 20, 4409-4411.
Shackelford, Rodney et al. “Desferrioxamine treatment increases the genomic stability ofAtaxia-telangiectasiacells” ScienceDirect, DNA Repair, vol. 2, Issu 9, Sep. 2003, pp. 971-981.
Simpson, Marty “Buck Researcher Links Iron to Parkinson's Disease” Buck Institute, Neuron Mar. 26, 2003.
Polla, Ada S., et al. “Iron as the malignant spirit in successful ageing” Ageing Research Reviews 2(2003) 25-37.
Young, Ian.S., et al. “The effects of desferrioxamine and ascorbate on oxidative stress in the streptozotocin diabetic rat” ScienceDirect, Free Radical Biology and Medicine, vol. 18, Issue 5, May 1995, pp. 833-840.
Kaur, Deepinder et al. “Genetic or Pharmacological Iron Chelation Prevents MPTP-Induced Neurotoxicity In Vivo” Science Direct, Neuron, vol. 37, Issue 6, Mar. 27, 2003, pp. 899-909.
Wong, Alice PhD, et al. “Oxidative Stress in Friedreich'sAtaxia: Mechanisms and Potential Therapy” Friedreich'sAtaxia(FRDA) NAF 1999.
Naughton, D.P., “Iron(III)-mediated intra-articular crystal deposition in arthritis: a therapeutic role for iron chelators” ScienceDirect, Medical Hypotheses, vol. 57, Issue 1, Jul. 2001, pp. 120-122.
Duffy, SJ, et al. “Iron chelation improves endothelial function in patients with coronary artery disease.” Entrez-Pub-Med, Abstract Jun. 12, 2001.
Kuperstein, Faina et al., “Pro-apoptotic signaling in neuronal cells following iron and amyloid beta peptide neurotoxicity” Journal of Neurochemistry, vol. 86, No. 1, 2003 114-125.
Cameron, NE., et al. “Neurovascular dysfunction in diabetic rates. Potential contribution of autoxidation and free radicals examined using transition metal chelating agents.” ABSTRACT Entrez-PubMed, J. Clin. Invest. Aug. 1995;96(2); 1159-63.
Buss, Joan L., et al. “The Role of Iron Chelation in Cancer Therapy” Abstract—Currently Medicinal Chemistry, vol. 10, No. 12, 2003.

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