Method for identifying metastatic sequences

Drug – bio-affecting and body treating compositions – Whole live micro-organism – cell – or virus containing – Genetically modified micro-organism – cell – or virus

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435375, 435 6, 4351723, 435 691, 935 62, 935 34, 800 2, C12N 500, C12N 1563, C12N 1579, A61K 4800

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057831823

ABSTRACT:
The invention relates to methods for the identification of metastatic sequences. Cells from a cell line or an animal tissue are treated to form a cell line predisposed to metastasis. Treated cells are implanted in an animal of a primary site and incubated for a period of time sufficient for the cells to proliferate and develop metastases at secondary sites. Expressed sequences from cells at the primary and secondary sites are amplified by differential display polymerase chain reaction and compared. Differentially expressed sequences are identical and can be cloned and sequenced. These sequences can be used as probes in the diagnosis of metastatic disorders, as probes to isolate metastatic sequences and as a therapeutic agent.

REFERENCES:
patent: 4317818 (1982-03-01), Benson et al.
patent: 4925835 (1990-05-01), Heston
patent: 5116615 (1992-05-01), Gokcen et al.
Xiong et al. "Human D-Type Cyclin," Cell, vol. 65: 691-699, May 17, 1991.
Manam et al. "Dose related changes in the profile of ras mutations in chemically induced CD-1 mouse liver tumors," Carcinogenesis, vol. 16 (5): 1113-1119, May 1995.
Blok, et al. "Isolation of cDNA's that are differentially expressed between androgen-dependent and androgen independent prostate carcinoma cells using differential display PCR," Prostate, vol. 26(4): 213-224, Apr. 1995.
Wu et al. "Identification of a human hepatocellular carcinoma-associated tumor suppressor gene by differential display polymerase chain reaction," Life Sciences, vol. 57(11): 1077-1085, Nov. 1995. Malignant Transformation Independent of Harvey ras Activation," J. of Investigative Dermatology, vol. 101 (4): 595-599, Oct. 1993.
Fingert et al. "In vivo model for differentiation therapy of leukemia and solid tumors," National Institutes of Health Publication, 84-2635, Serono Symposia Publications from Rven Press, pp. 277-286, 1984.
Taber's Cyclopedic Medical Dictionary, F.A. Davis Company, Philidelphia, PA, edited by Vardara et al., 1993.
Gudas, "Retinoids, Retinoid-responsive Genes, Cell Differentiation, and Cancer"; Cell Growth & Differentiation, vol. 3, pp. 655-662, Sep. 1992.
Mokulis, et al., "Screening for Prostate Cancer: Pros, Cons, and Reality"; Cancer Control, pp. 15-21, Jan./Feb. 1995.
Merz, et al., "Elevated Transforming Growth Factor-.beta.1 and .beta.3 mRNA Levels are Associated with ras+myc-Induced Carcinomas in Reconstituted Mouse Prostate: Evidenced for a Paracrine Role during Progression", Molecular Endocrinology, vol. 5, No. 4, (1991) pp. 503-513.
Poster Session Abstracts; First SPORE Investigators' Meeting, "The Role of Retinoids in Prostate Cancer Chemoprevention" Jul. 18-20, 1993, p. 30.
Slawin, et al., "Dietary Fenretinide, a Synthetic Retinoid, Decreases the Tumor Incidence and the Tumor Mass of ras+myc-induced Carcinomas in the Mouse Prostate Reconstitution Model System", Cancer Research, vol. 53, pp. 4461-4465, Oct. 1, 1993.
Thompson, et al., "Transgenic Models for the Study of Prostate Cancer",(Supplement) Cancer, vol. 71, No. 3, Feb. 1, 1993, pp. 1165-1171.
Donehower, et al, "Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours", Articles, Nature, vol. 356, Mar. 19, 1992, pp. 215-221.
Thompson, et al., "Loss of p53 function leads to metastasis in ras+myc-initiated mouse prostate cancer", Oncogene (1995) vol. 10, pp. 869-879.
Macoska, et al., "Loss of the 17p Chromosomal Region in a Metastatic Carcinoma of the Prostate", The Journal of Urology, vol. 147, Apr. 1992, pp. 1142-1146.
Taylor, et al., "Evidence for synergistic interactions between ras, myc and a mutant form of p53 in cellular transformation and tumor dissemination", Oncogene, Feb. 10, 1992, pp. 1383-1390.
Hall, et al., "Adenylate Kinase: An Oncodevelopmental Marker in an Animal Model for Human Prostatic Cancer", Clinical Chemistry, vol. 31, No. 10, (1985), pp. 1689-1691.
Thompson, et al., Multistage Carcinogenesis Induced by ras and myc Oncogenes in a Reconstituted Organ, Cell, vol. 56, pp. 917-930, Mar. 24, 1989.
Slawin, et al., American Urological Association, Inc., Annual Meeting--San Antonio, Oct. 1, 1992, Dietary Retinoids Decrease the Incidence and Increase Lymphocytic Infiltration of ras+myc Induced Carcinomas in the Mouse Prostate Reconstitution Model System.
Thompson, et al., "Transforming Growth Factor .beta.1 as a Biomarker for Prostate Cancer", Journal of Cellular Biochemistry, Supplement 16H: pp. 54-61 (1992).
Thompson, et al., "Genetic Predisposition and Mesenchymal-Epithelial Interactions in ras+myc-Induced Carcinogenesis in Reconstituted Mouse Prostate" Molecular Carcinogenesis, vol. 7, pp. 165-179 (1993).
Bookstein et al., "p53 Is Mutated in a Subset of Advanced-Stage Prostate Cancers.sup.1 ", Cancer, vol. 53, pp. 3369-3373, Jul. 19, 1993.
Carter, et al. "Prediction of Metastatic Potential in an Animal Model of Prostate Cancer: Flow Cytometric Quantification of Cell Surface Charge", The Journal of Urology, vol. 142, pp. 1338-1341, Nov. 1989.
Fox, et al., "p53 And c-myc Expression in Stage A1 Prostatic Adenocarcinoma: Useful Prognostic Determinants?", The Journal of Urology, vol. 150, pp. 490-494, Aug. 1993.
Einstein, "Hormonal Therapy for Prostate Cancer--When to Use It", Cancer Control, Jan./Feb. 1995, pp. 32-36.
Thompson, et al., "Loss of p53 Function Leads to Metastasis in ras+myc-Initiated Mouse Prostate Cancer", Abstract for Fogarty International Meeting, Jun. 26-28, 1995.
International Search Report, completed May 30, 1997.
Welch, Danny R., et al. "Transforming growth factor .beta. stimulates mammary adenocarcinoma cell invasion and metastatic potential", Proc. Natl. Acad. Sci. USA, vol. 87, pp. 7678-7682. Oct. 1990.
Thompson, Timothy C., et al. "Multistage Carcinogenesis Induced by ras and myc Oncogenes in a Reconstituted Organ", Cell, vol. 56, pp. 917-930. Mar. 24, 1990.
Liang, Peng, et al. "Differential Display and Cloning of Messenger RNAs from Human Breast Cancer versus Mammary Epithelial Cells", Cancer Research, 52, pp. 6966-6968. Dec. 15, 1992.
Proceedings of the American Association for Cancer Research, vol. 36, p. 266 #1589. Mar. 1995.
Liang, Peng, et al. "Differential Display of Eukaryotic Messenger RNA by Means of the Polymerase Chain Reaction", Science, vol. 257, pp. 967-971. Aug. 14, 1992.
Wood, David P., Jr., et al.. "Sensitivity of Immunohistochemistry and Polymerase Chain Reaction in Detecting Prostate Cancer Cells in Bone Marrow", The Journal of Histochemistry and Cytochemistry, vol. 42, No. 4, pp. 505-511. 1994.

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