Human genes of chromosome 11q13.3

Chemistry: natural resins or derivatives; peptides or proteins; – Proteins – i.e. – more than 100 amino acid residues

Reexamination Certificate

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C435S069100, C435S320100, C435S325000, C435S455000, C530S351000, C536S023100, C536S023500

Reexamination Certificate

active

07151161

ABSTRACT:
This invention relates to isolated nucleic acids comprising novel genes of a 20 cM region of human chromosome 11q13.3 and the proteins encoded by these genes. Expression vectors and host cells containing these genes or fragments thereof, as well as antibodies to the proteins encoded by these nucleic acids are also included herein.

REFERENCES:
patent: 5691153 (1997-11-01), Recker et al.
patent: 98/39448 (1998-09-01), None
Altier et al Neuropharmacol. 40:1050-1057, 2001.
ASBMR 18th Annual Meeting, Supplement 1, S661, vol. 11, 1996 (American Society of Bone & Mineral Research, U.S.A.).
Am. J. Hum. Genet., “Linkage of a Gene Causing High Bone Mass to Human Chromosome 11 (11q12-13)” Mark K. Johnson et al., 60:1326-1332, pp. 1326-1332, 1997 (University of Chicago Press, U.S.A.).
Am. J. Hum. Genet., “Fine Mapping of the Diabetes-Susceptibility Locus, IDDM4, on Chromosome 11q13” Yusuke Nakagawa et al., 63:547-556, pp. 547-556, 1998 (University of Chicago Press, U.S.A.).
Proc. Natl. Acad.. Sci. USA, “Ion Channel Genes and Human Neurological Disease: Recent Progress, Prospects, and Challenges”, Edward C. Cooper et al., vol. 96, pp. 4759-4766, Apr. 1999 (National Academy of Science, U.S.A.).
Journal of Bone and Mineral Research, “Linkage of a QTL Contributing to Normal Variation in Bone Mineral Density to Chromosome 11q12-13”, D.L. Koller et al., vol. 12, No. 12, pp. 1903-1908, 1998, (American Society of Bone & Mineral Research, U.S.A.).

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