Chemistry: molecular biology and microbiology – Animal cell – per se ; composition thereof; process of...
Reexamination Certificate
2003-06-16
2009-06-30
Hutson, Richard G (Department: 1652)
Chemistry: molecular biology and microbiology
Animal cell, per se ; composition thereof; process of...
C435S320100, C435S069100, C435S252300, C435S254110, C536S023100, C536S023200, C536S024300
Reexamination Certificate
active
07553659
ABSTRACT:
Nucleic acid sequences encoding chromodomain helicase DNA-binding (CHD) proteins, CHD proteins, and antibodies thereto and methods of use thereof are disclosed.
REFERENCES:
Ngo et al. in The Protein Folding Problem and Tertiary Structure Prediction, 1994, Merz et al. (ed.), Birkhauser, Boston, MA, pp. 433 and 492-495.
Thompson et al., GenEMBL database Accession No. AF425231.1, Mar. 28, 2002.
Ahringer, NuRD and SIN3 histone deacetylase complexes in development, TIG (2000) V. 16(8), pp. 351-355.
Ayer et al., Mad-Max Transcriptional Repression is Mediated by Ternary Complex Formation with Mammalian Homologs of Yeast Repressor SIN3, Cell (1995) V. 80, pp. 767-776.
Bentley et al., The physical maps for sequencing human chromosomes 1, 6, 9, 10, 13, 20, and X, Nature (2001), V. 409, pp. 942-943.
Brehm et al., Retinoblastoma protein recruits histone deacetylase to repress transcription, Nature (1998) V. 391(5), pp. 597-601.
Brehm et al., The E7 oncoprotein associates with Mi2 and histone deacetylase activity to promote cell growth, The EMBO Journal (1999) V. 18(9), pp. 2449-2458.
Brodeur et al., Biology and Genetics of Human Neuroblastomas, Journal of Pediatric Hematology/Oncology (1997), V. 19(2), pp. 93-101.
Caron et al., Evidence for two tumour suppressor loci on chromosomal bands 1p35-36 involved in neuroblastoma: one probably imprinted, another associated with N-myc amplification, Human Molecular Genetics (1995) V. 4(4), pp. 535-539.
Cohn et al., Prolonged N-mycprotein half-life in a neuroblastoma cell line lacking N-mycamplification, Oncogene (1990) V. 5, pp. 1821-1827.
Delmas et al., A mammalian DNA-binding protein that contains a chromodomain and an SNF2/SW12-like helicase domain, PNAS (1993), V. 90, pp. 2414-2418.
Eggert et al., Direct Effects of TrkA Expression in Neuroblastoma Cell Lines With or WithoutMYCNAmplification, Medical and Pediatric Oncology (2000), V. 35, pp. 623-627.
Eggert et al., Molecular dissection of TrkA signal transduction pathways mediating differentiation in human neuroblastoma cells, Oncogene (2000), V. 19, pp. 2043-2051.
Ge et al., Molecular Analysis of a Major Antigenic Region of the 240-kD Protein of Mi-2 Autoantigen, J. Clin. Invest. (1995), V. 96, pp. 1730-1737.
Grant et al., Yeast Gcn5 functions in two multisubunit complexes to acetylate nucleosomal histones: characterization of an Ada complex and the SAGA (Spt/Ada) complex, Genes and Development (1997), V. 11, pp. 1640-1650.
Grignani et al., Fusion proteins of the retinoic acid receptor-α recruit histone deacetylase in promyelocytic leukaemia, Nature (1998) V. 391, pp. 815-818.
Harper et al., Sin3 corepressor function in Myc-induced transcription and transformation, PNAS (1996), V. 93, pp. 8536-8540.
Hogarty et al., Identification of a 1-Megabase Consensus Region of Deletion at 1p36.3 in Pirmary Neuroblastomas, Medical and Pediatric Oncology (2000), 35, pp. 512-515.
Kasten et al., SIN3-Dependent Transcriptional Repression by Interaction with the Mad1 DNA-Binding Protein, Molecular and Cellular Biology (1996), V. 16(8), pp. 4215-4221.
Kehle et al., dMi-2, a Hunchback-Interacting Protein that Functions inPolycombRepression, Science (1998), V. 282, pp. 1897-1900.
Kitamura et al., Histone deacetylase inhibitor but not arsenic trioxide differentiates acute promyelocytic leukaemia cells with t(11;17) in combination with all-transretinoic acid, British Journal of Haematology (2000), V. 108, pp. 696-702.
Kozak, Do the 5′ untranslated domains of Human cDNAs Challenge the Rules for Initiation of Translation (or Is It Vice Versa)?, Genomics (2000), V. 70, pp. 396-406.
Lin et al., Role of histone deacetylase complex in acute promyelocytic leukaemia, Nature (1998), V. 391 pp. 811-814.
Lipshutz et al., High density synthetic oligonucleotide arrays, nature genetics supplement, (1999), V. 21 pp. 20-24.
Luo et al., Rb interacts with Histone Deacetylase to Repress Transcription, (1998), V. 92, pp. 463-473.
Magnaghi-Jaulin et al., Retinoblastoma protein represses transcription by recruiting a histone deacetylase, Nature (1998), V. 391, pp. 601-605.
Maris et al., Cloning, Chromosomal Localization, Physical Mapping, and Genomic Characterization of HKR3, Genomics (1996), V. 35, pp. 289-298.
Maris et al., Comprehensive Analysis of Chromosome 1p Deletions in Neuroblastoma, Medical and Pediatric Oncology (2001), V. 36, pp. 32-36.
Müller et al., Transcriptional silencing by the Polycomb protein inDrosphiliaembryos , The EMBO Journal (1995), pp. 1209-1220.
Owen-Hughes et al., Analysis of Nucleosome Disruption by ATP-Driven Chromatin Remodeling Complexes, Methods in Molecular Biology V. 119, pp. 319-331.
Owen-Hughes et al., Remodeling the chromatin structure of a nucleosome array by transcription factor-targetedtrans-displacement of histones, The EMBO Journal (1996), V. 15(17), pp. 4702-4712.
Reese et al., Genome Annotation Assessment inDrosophilia melanogaster, Genome Research (2000), V. 10, pp. 483-501.
Reynolds et al., Biological Classification of Cell Lines Derived from Human Extra-cranial Neural Tumors, Advances in Neuroblastoma Research 2, (1988), pp. 291-306.
Rundlett et al., HDA1 and RPD3 are members of distinct yeast histone deacetylase complexes that regulate silencing and transcription, PNAS, (1996) V. 93, pp. 14503-14508.
Schmidt et al., Molecular Association between ATR and Two Components of the Nucleosome Remodeling and Deacetylating Complex, HDAC2 and CHD4+, Biochemistry, (1999), V. 38, pp. 14711-14717.
Schreiber-Agus et al., An Amino-Terminal Domain of Mxi1 Mediates Anti-Myc Oncogenic Activity and Interacts with a Homolog of the Yeast Transcription Repressor SIN3, Cell (1995), V. 80, pp. 777-786.
Schreiber-Agus et al., Repression by the Mad(Mxi1)-Sin3 complex, BioEssays, (1998), V. 20, pp. 808-818.
Schuster et al., CHD5 defines a new subfamily of chromodomain-SW12/SNF2-like helicases, Mammalian Genome, (2002), V. 13, pp. 117-119.
Seelig et al., The Major Dematomyositis-specific Mi-2 Autoantigen is a Presumed Helicase Involved in Transcriptional Activation, Arthritis and Rheumatism, (1995), V. 38 N. 10, pp. 1389-1399.
Slavotinek et al., Monosomy 1p36, J. Med. Genet., (1999), V. 36, pp. 657-663.
Struhl, Histone acetylation and transcriptional regulatory mechanisms, Genes and Development, (1998), V. 12, pp. 599-606.
Surapureddi et al., Identification of a transcriptionally active peroxisome proliferator-activated receptor α-interacting cofactor complex in rat liver and characterization of PRIC285 as a cofactor, PNAS (2002), V. 99, N. 18, pp. 11836-11841.
Sudarsanam et al., The Swi/Snf family nucleosome-remodeling complexes and transcriptional control, TIG (2000), V. 16, N. 8, pp. 345-350.
Tatusova et al., BLAST 2 Sequences, a new tool for comparing protein and nucleotide sequences, FEMS Microbiology Letters 174 (1999), pp. 247-250.
Thompson et al., Homozygous Deletion of CDKN2A (p16INK4a/p14ARF) but not within 1p36 or at Other Tumor Supressor Loci in Neuroblastoma, Cancer Research (2001), V. 61, pp. 679-686.
Thompson et al., CHD5, a new member of the chromodomain gene family, is preferentially expressed in the nervous system, Oncogene, (2003), V. 22, N. 7, pp. 1002-1011.
Tong et al., Chromatin deacetylation by an ATP-dependent nucleosome remodeling complex, Nature (1998), V. 395, pp. 917-921.
Wang et al., Architectural DNA binding by a high-mobility-group/kinesin-like subunit in mammalian SWI/SNF-related complexes, PNAS (1998), V. 95 pp. 492-498.
White et al., Molecular Analysis of the Region of Distal 1p Commonly Deleted in Neuroblastoma, European Journal of Cancer (1997), V. 33, N. 12, pp. 1957-1961.
White et al, Detailed Molecular Analysis of 1p36 in Neuroblastoma, Medical and Pediatric Oncology, (2001), V. 36, pp. 36-41.
Wong et al., BRG1, a Component of the SWI-SNF Complex, Is Mutated in Multiple Human Tumor Cell Lines, Cancer Research (2000), V. 60, pp. 6171-6177.
Woodage et al., Characterization of the CHD family of proteins, PNAS (1997), V. 94, pp. 11472-11477.
Wu et al., Molecular refinement of the 1p36 deletion syndrome reveals size diversity and a preponderance of maternally
Brodeur Garrett M.
White Peter S.
Dann Dorfman Herrell & Skillman
Hutson Richard G
Netter Robert C.
Rigaut Kathleen D.
The Children's Hospital Of Philadelphia
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