Human Gil-19/AE289 polynucleotides

Organic compounds -- part of the class 532-570 series – Organic compounds – Carbohydrates or derivatives

Reexamination Certificate

Rate now

  [ 0.00 ] – not rated yet Voters 0   Comments 0

Details

C435S069100, C435S252300, C435S320100, C435S325000, C435S471000

Reexamination Certificate

active

09561811

ABSTRACT:
Novel human GIL-19/AE289 protein is disclosed which shows a high degree of homology to interleukin-10 (IL-10). Polynucleotides encoding such protein are also enclosed.

REFERENCES:
patent: 5350836 (1994-09-01), Kopchick et al.
patent: 5536637 (1996-07-01), Jacobs
patent: 6274710 (2001-08-01), Dumoutier et al.
patent: 6331613 (2001-12-01), Dumoutier et al.
patent: 6359117 (2002-03-01), Dumoutier et al.
patent: 6551799 (2003-04-01), Gurney et al.
patent: 2001/0024652 (2001-09-01), Dumoutier et al.
patent: 2002/0012669 (2002-01-01), Presnell et al.
patent: 2002/0102723 (2002-08-01), Gurney et al.
patent: 2002/0187523 (2002-12-01), Tang et al.
patent: 2003/0012788 (2003-01-01), Renauld et al.
patent: 2003/0170823 (2003-09-01), Presnell et al.
patent: 2004/0023341 (2004-02-01), Wenfeng et al.
patent: 2004/0110189 (2004-06-01), Dumoutier et al.
patent: 2004/0152125 (2004-08-01), Presnell et al.
patent: 2004/0180399 (2004-09-01), Renauld et al.
patent: WO-94/01548 (1994-01-01), None
patent: WO99/61617 (1999-12-01), None
patent: WO 2000/24758 (2000-05-01), None
patent: WO 00/70049 (2000-11-01), None
patent: WO 00/73457 (2000-12-01), None
patent: WO 00/77037 (2000-12-01), None
patent: WO 01/46422 (2001-06-01), None
patent: WO 02/10393 (2002-02-01), None
patent: WO2002/16611 (2002-02-01), None
Mahairas G, et al. Database EST. Accession No. AQ104025. Aug. 28, 1998.
Waterston R, et al. Database GenEmbl. Accession No. AC006734. Feb. 25, 1999.
Wilson R, et al. J. Mol. Biol. 261:155-172, 1996.
Bork et al. Trends in Genetics 12:425-427, 1996.
Vukicevic et al. PNAS USA 93:9021-9026, 1996.
Massague J. Cell 49:437-8, 1987.
Pilbeam et al. Bone 14:717-720, 1993.
Skolnick et al. Trends in Biotech. 18:34-39, 2000.
Bork P. Genome Research 10:398-400, 2000.
Doerks et al. Trends in Genetics 14:248-250, 1998.
Smith et al. Nature Biotechnology 15:1222-1223, 1997.
Brenner SE. Trends in Genetics 15:132-133, 1999.
Dumoutier et al. (2000), “Cloning and characterization of IL-10-related T cell-derived inducible factor (IL-TIF), a novel cytokine structurally related to IL-10 and inducible by IL-9,”J. of Immunol.164:1814-1819.
Syrbe et al, (1999) Springer Seminars in Immunopathology, 21:263-85.
Aoki, I. et al. “Comparison of the amino acid and nucleotide sequences between human and two guinea pig major basic proteins,”FEBS Lett.Apr. 22, 1991;282(1):56-60.
Dumoutier, L. et al., “Human interleukin-10-related T cell-derived inducible factor: molecular cloning and functional characterization as an hepatocyte-stimulating factor,”PNAS2000 97(18):10144-9.
Dumoutier, L. et al., “IL-TIF/IL-21: genomic organization and mapping of the human and mouse genes,”Genes Immun.2000;1:488-494.
Ozaki, T et al., “Developmental regulation of transcription of a novel prespore-specific gene (Dp87) inDictyostelium discoideum,” Development.Apr. 1993;1.17(4):1299-308.
Sambrook, J. et al.Molecular Cloning. A laboratory manual, 2d ed. Cold Spring Harbor Laboratory Press, 1989, Ch. 17.
Xie, M.H. et al., “Interleukin (IL)-22, a novel human cytokine that signals through the interferon receptor-related proteins CRF2-4 and IL-22R,”J. Biol. Chem.Oct. 6, 2000;275(40):31335-9.
Dumoutier, L. et al: “Cloning and Characterization of IL-10-Related T Cell-Derived Inducible Factor (IL-TIF), A Novel Cytokine Structurally Related to IL-10 and Inducible By IL-9” Journal of Immunology, Blackwell Scientific Publications, GB, vol. 164, 2000, pp. 1814-1819.
Dumoutier, L. et al: “Human Interleukin-10-Related T Cell-Derived Inducible Factor: Molecular Cloning and Functional Characterization as an Hepatocyte-Stimulating Factor” Proceedings of the National Acadamy of Sciences of USA, National Acadamy of Science. Washington, US, vol. 97, No. 18, Aug. 29, 2000, pp. 10144-10149.
Xie, M-H et al: “Interleukin (IL)-22, a novel human cytokine that signals through the interferon receptor-related proteins CRF2-4 and IL-22R” Journal of Biological Chemistry, American Society of Biological Chemists, Baltimore, MD, US, vol. 275, No. 40, Oct. 6, 2000, pp. 31335-31339.
Kotenko, Sergei V. et al: “Identification of the functional interleukin-22 (IL-22) receptor complex. The IL-10R2 chain (IL-10Rbeta) is a common chain of both the IL-10 and IL-22 (IL-10-related T cell-derived inducible factor, IL-TIF) receptor complexes” Journal of Biological Chemistry, American Society of Biological Chemists, Baltimore, MD, US, vol. 276, No. 4, Jan. 26, 2001, pp. 2725-2732.
Dumoutier, L. et al: “IL-TIF induces acute phase reactant production by hepatocytes through IL-10Rbeta.” Immunology Letters, vol. 73, No. 2-3 Sep. 2000, p. 261.
Lambert, A. et al: “Novel cytokine IL-22 administrated by adenovirus vector or as recombinant purified protein induces acute-phase responses and renal tubular basophilia in female C57BL/6 mice.” Toxicologic Pathology, vol. 29, No. 6, Nov. 2001, p. 712.
R&D Systems, Catalog NR, AF582, XP002307633, “Anti-Mouse IL-22 Antibody”, Aug. 22, 2002.
Kotenko, Sergei, “The Family of IL-10-Related Cytokines and Receptors: Related, But to What Extent?”, Cytokine and Growth Factor Reviews, vol. 13, No. 3, Jun. 2002, pp. 223-240.
Radaeva, Svetlana, et al, “Interleukin 22 (IL-22) Plays a Protective Role in T Cell-Mediated Murine Hepatitis: IL-22 is a Survival Factor for Hepatocytes via STAT3 Activation”, Hepatology, vol. 39, No. 5, May 2004, pp. 1332-1342.
Resmini, Christine, et al, “An Anti-Murine IL-22 Monoclonal Antibody Decreases Disease Severity in a Murine Model of Collagen Induced Arthritis”, European Cytokine Network, vol. 14, No. Supplement 3, Sep. 2003, p. 129 and Annual Meeting of the International Cytokine Soceity; Dublin, Ireland, Sept 20-24, 2003, ISSN: 1148-5493.
Li, J., et al., “Temporal Associations Between Interleukin 22 and the Extracellular Domains of IL-22R and IL10R2”, International Immunopharmacology, Elsevier, Amsterdam, NL, vol. 4, No. 5, May 2004, pp. 693-708.
Dumoutier, L et al: “Human Interleukin-10-Related T Cell-Derived Inducible Factor: Molecular Cloning and Functional Characterization as an Hepatocyte-Stimulating Factor” Proceedings of the National Academy of Sciences of USA, National Acadamy of Science. Washington, US vol. 97, No. 18, Aug. 29, 2000, pp. 10144-10149.
Dumoutier, L., et al. GenBank Accession No. NM—016971 forMus musculusinterleukin 10-related T cell-derived inducible factor (Iltif). Jun. 8, 2000.
Ozaki, T., et al. GenBank Accession No. D13973 forDictyostelium discoideumDNA for Dp87 protein, 1993. Feb. 1, 2000.
Aoki, I., et al. GenBank Accession No. P35709 for Eosinophil granule major basic protein 2 precursor (mbp-2). May 30, 2000.
Mahairas, G.G., et al. GenBank Accession No. AQ104025 for HS—3108—B1—C01—T7 CIT Approved Human Genomic Sperm Library DHomo sapiensgenomic clone Plate=3108 Col=1 Row=F. Aug. 28, 1998.
Xie, M., et al. GenBank Accession No. AF279437 forHomo sapiensinterleukin 22 (IL22). Oct. 9, 2000.
Dumoutier, L., et al. GenBank Accession No. AJ294727 forMus musculusILTIFa gene for IL TIF alpha protein (IL-21), exons 1a-5. Dec. 21, 2000.
Dumoutier, L., et al. GenBank Accession No. NP—065386 for Interleukin 22; interleukin 21; IL-10-related T-cell-derived inducible factor (Homo sapiens). Nov. 2, 2000.

LandOfFree

Say what you really think

Search LandOfFree.com for the USA inventors and patents. Rate them and share your experience with other people.

Rating

Human Gil-19/AE289 polynucleotides does not yet have a rating. At this time, there are no reviews or comments for this patent.

If you have personal experience with Human Gil-19/AE289 polynucleotides, we encourage you to share that experience with our LandOfFree.com community. Your opinion is very important and Human Gil-19/AE289 polynucleotides will most certainly appreciate the feedback.

Rate now

     

Profile ID: LFUS-PAI-O-3895646

  Search
All data on this website is collected from public sources. Our data reflects the most accurate information available at the time of publication.