Hypoglycemics

Drug – bio-affecting and body treating compositions – Designated organic active ingredient containing – Peptide containing doai

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514 13, 514866, A61K 3702, A61K 3743

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active

052602753

ABSTRACT:
Non-insulin dependent, or type 2, diabetes mellitus in a patient is treated by administering to the patient a hypoglycemic agent that enhances plasma concentrations of amylin and a therapeutically effective amount of an amylin antagonist. Hypoglycemic agents which enhance plasma concentrations of amylin can be sulfonylureas such as glibenclamide and tolbutamide. Amylin antagonists can be amylin 8-37 and CGRP 8-37. Administration of the amylin antagonist in conjunction with the hypoglycemic agent also enhances the blood glucose lowering effects of the hypoglycemic agent.

REFERENCES:
Leighton et al, "Pancreatic amylin and CGRP cause resistance to insulin . . . ", Nature 335:632-35 (1988).
Mitsukawa et al, "Islet Amyloid Polypeptide Response to Glucose, Insulin, . . . ", Diabetes, vol. 39, May 1990, pp. 639-642.
Moline et al, "Induction of Insulin Resistance In Vivo by Amylin and CRP", Diabetes, vol. 39, Feb. 1990, pp. 260-265.
Westermark et al., Proc. Natl. Acad. Sci. U.S.A. 84:3881-85 (1987).
Cooper et al., Proc. Natl. Acad. Sci. U.S.A. 84:8628-32 (1987).
Joslin's Diabetes Mellitus, Chapter 21 (12th ed. 1985).
Goodman and Gilman's: The Pharmacological Basis of Therapeutics, p. 1504 (7th ed. 1988).
Nishi et al., Journal of Biological Chemistry 265:4173-76 (1990).
Clark, Diab. Med. 6:561-67 (1989).
Cooper et al., Biochim. Biophy. Acta 1014:247-58 (1989).
Cooper et al., Diabetes 1988, pp. 493-496, Larkins, Zimmet, and Chisholm (Eds.), (Elsevier Science Publishers B. V. 1989).
Cooper et al., Progress in Growth Factor Research 1:99-105 (1989);
Johnson et al., New England Journal of Medicine 321:513-18 (1989).

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